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Although specific guidelines are not available, dosage reduction may be necessary in patients with hepatic dysfunction. Food may reduce the rate, but not the extent of absorption. Biliary excretion of unchanged ondansetron vademecum is a minor elimination pathway, and enterohepatic recycling of oxaprozin is insignificant. NSAIDs may mask the signs of infection such as fever or pain in patients with bone marrow suppression.
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Due to their effect on prostaglandin synthesis, oxaprozin and other NSAIDs may worsen fluid retention by the kidneys and thus, increase blood pressure in susceptible individuals.
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Preclinical data suggest agents that inhibit prostaglandin synthesis such as NSAIDs could decrease the efficacy of porfimer or verteporfin photodynamic therapy. Renal dysfunction alters oxaprozin binding and reduces the clearance of unbound drug.
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